Lipopolysaccharide-Induced Acute Lung Injury Is Associated with Increased Ran-Binding Protein in Microtubule-Organizing Center (RanBPM) Molecule Expression and Mitochondria-Mediated Apoptosis Signaling Pathway in a Mouse Model

Xiaojing Lv; Xiaomin Lu; Jiping Zhu; Qian Wang

doi:10.12659/MSM.923172

18 July 2020 : Animal Research

Lipopolysaccharide-Induced Acute Lung Injury Is Associated with Increased Ran-Binding Protein in Microtubule-Organizing Center (RanBPM) Molecule Expression and Mitochondria-Mediated Apoptosis Signaling Pathway in a Mouse Model

Xiaojing Lv^12BCDEF, Xiaomin Lu^12BCDEF, Jiping Zhu^12ABDEG*, Qian Wang^12BCF

DOI: 10.12659/MSM.923172

Med Sci Monit 2020; 26:e923172

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Figure 1 Pathological changes of inflammations in lung tissues for the ALI mouse models in different groups according to HE staining. (A) HE staining images. (B) Statistical analysis of histological scores for HE-stained images. Magnification, 100×. * p<0.05 vs. Control group.

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Lipopolysaccharide-Induced Acute Lung Injury Is Associated with Increased Ran-Binding Protein in Microtubule-Organizing Center (RanBPM) Molecule Expression and Mitochondria-Mediated Apoptosis Signaling Pathway in a Mouse Model

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