miR-1283 Contributes to Endoplasmic Reticulum Stress in the Development of Hypertension Through the Activating Transcription Factor-4 (ATF4)/C/EBP-Homologous Protein (CHOP) Signaling Pathway

Weihao Chen; Tianhao Liu; Qiuer Liang; Xudong Chen; Wencong Tao; Meixia Fang; Ya Xiao; Liguo Chen

doi:10.12659/MSM.930552

29 April 2021 : Animal Research

miR-1283 Contributes to Endoplasmic Reticulum Stress in the Development of Hypertension Through the Activating Transcription Factor-4 (ATF4)/C/EBP-Homologous Protein (CHOP) Signaling Pathway

Weihao Chen^1ABC, Tianhao Liu^1EF*, Qiuer Liang^1F, Xudong Chen^1BC, Wencong Tao^1B, Meixia Fang^1AE, Ya Xiao^1AE, Liguo Chen^1AEG

DOI: 10.12659/MSM.930552

Med Sci Monit 2021; 27:e930552

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Figure 1 The illustration of miR-1283 targeting of activating transcription factor 4 (ATF4) participating in the development of salt-induced hypertension via the ATF4/CHOP signaling pathway. Control – control group; mimic – miR-1283 mimic group; inhibitor – miR-1283 inhibitor group; GSK – GSK2656157; mimic control – miR-1283 mimic negative control group; inhibitor control – miR-1283 inhibitor negative control group.

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miR-1283 Contributes to Endoplasmic Reticulum Stress in the Development of Hypertension Through the Activating Transcription Factor-4 (ATF4)/C/EBP-Homologous Protein (CHOP) Signaling Pathway

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